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Sorry das ich mich missverständlich ausgedrückt habe!
Viele Wege führen nach Rom!
Für Bart ist rifa und atzi oder Tavanic Mittel der Wahl.
Bei Mischinfektionen ist Cotrimoxazol super, weil es sowohl Bart, Babs und CPN erwischt.
http://www.lymebook.com/antibiotic-treatment-for-babesia-bartonella-ehrlichia-co-infections
Colibri Keime sind ganz seltene Infektionen. Wie ein Colibri halt , selten azutreffen.
Wenn du dir nicht sicher bist wer der Verursacher allen Übels ist , würde ich Cotrim forte + Atzi + Artemisinin nehmen.
Damit triffst du CPN, Bart, und Protozooen…..
VG
Popo
und gib bei search mal artemisinin ein….
Das wird empfohlen….
http://flash.lymenet.org/scripts/ultimatebb.cgi/ubb/search/search_forum/1
Cotrimoxazol forte (2×1 am Tag)ausprobieren. (Covers >Bart and Babs) In Verbindung mit Roxy auch als gasser therapie bekannt und für CPN ebenfalls super.
Im CPNhelp Forum auch als roxy/Bactrim Combo zu finden…..
Mit Cotrim erwischt mann viele Colibri Keime!
Mir hilft es am meisten…..
Bartonellen tests sind genauso wie babesien tests fürn A…..!
Burrascano(Lyme Pabst) sagt aufgrund von Klinik behandeln und nicht von Titern. Diese sind bei vielen negativ….
Die Viren würde ich nicht checken, aber mycoplasma , Ehrlichia und die anderen Bakterien…
Lg
Bartonella
Explanation: Bartonella spp. bacterium
Symptoms: Fever, chills, headache and severe pain in the tibia, weight loss, sore throat, papular or angiomatous rash,burning feet
Treatment: Erythromycin, plus a Fluoroquinolone or RifampinDR. BURRASCANO’S 2002 … LYME, BARTONELLA, BABS, & ERLICHIA SYMPTOMS LIST
http://flash.lymenet.org/ubb/ultimatebb.php?ubb=get_topic;f=1;t=062695
Bartonellosis symptoms from fuzzyslippers 2.21.09
***********************Common symptoms of bartonellosis include:
___Fatigue (often with agitation, unlike Lyme disease, which is more exhaustion)
___Low grade fevers, especially morning and/or late afternoon, often associated with feelings of „coming down with the flu or a virus“
___Sweats, often morning or late afternoon (sometimes at night) – often described as „thick“ or „sticky“ in nature
___Headaches, especially frontal (often confused with sinus) or on top of head
___Eye symptoms including episodes of blurred vision, red eyes, dry eyes
___Ringing in the ears (tinnitus) and sometimes hearing problems (decreased or even increased sensitivity – so-called hyperacusis)
___Sore throats (recurring)
___Swollen glands, especially neck and under arms
___Anxiety and worry attacks; others perceive as „very anxious“
___Episodes of confusion and disorientation that are usually transient (and very scary); often can be seizure-like in nature
___Poor sleep (especially difficulty falling asleep); poor sleep quality
___Joint pain and stiffness (often both Left and Right sides as opposed to Lyme which is often on one side only with pain and stiffness that changes locations)
___Muscle pains especially the calves; may be twitching and cramping also
___Foot pain, more in the morning involving the heels or soles of the feet (sometimes misdiagnosed as plantar fasciitis)
___Nerve irritation symptoms which can be described as burning, vibrating, numb, shooting, etc.
___Tremors and/or muscle twitching
___Heart palpitations and strange chest pains
___Episodes of breathlessness
___Strange rashes recurring on the body often, red stretch marks, and peculiar tender lumps and nodules along the sides of the legs or arms, spider veins
___Gastrointestinal symptoms, abdominal pain and acid reflux
___Shin bone pain and tenderness
Bartonella is a bacterium that causes illness, the most commonly known of which is a disease called „Cat Scratch Fever.“
Thousands of known cases of Bartonella occur in the U.S. each Year, with the vast majority of known cases due to bites from fleas that infest cats or infected dogs (may also occur directly from bites and scratches from infected dogs or cats).
Bartonella can also be transmitted by ticks that transmit Lyme Disease.
In fact, in a study published recently, deer ticks from New Jersey had a higher prevalence of Bartonella organisms than of Lyme organisms.
It is unclear whether the organism that we see transmitted along with Lyme disease is actually a Bartonella species (such as B. henselae or B. quintana) or is „Bartonella-Like Organism“ (BLO) that is yet to be fully identified.
While BLO has features similar to organisms in the Bartonella family, it also has features slimiar to the Mycoplasma and the Francisella (causes tularemia) families.
editing; added part i overlooked to add!
Babesiosis
As with other co-infections, there is a lot of overlap of symptoms between Lyme disease and Babesiosis. An accumulation of the following signs and symptoms probably warrant testing and/or treatment of Babesiosis:
___Chills
___Fatigue and often excessive sleepiness
___High fever at onset of illness
___Night sweats that are often drenching and profuse
___Severe muscle pains, especially the large muscles of the legs (quads, buttocks, etc.)
___Neurological symptoms often described as „dizzy, tipsy, and spaciness,“ similar to a sensation of „floating“ or „walking off the top of a mountain onto a cloud“
___Depression
___Episodes of breathlessness, „air hunger“, and/or cough
___Decreased appetite and/or nausea
___Spleen and/or liver enlargement
___Abnormal labs (low white blood count, low platelet counts, mild elevation of liver enzymes, and elevated sed rate)
___Headaches (migraine-like, persistent, and especially involving the back of the head and upper neck areas)
___Joint pain (more common with Lyme and Bartonella)
___anxiety/panic (more common with Bartonella)
___Lymph gland swelling (more common with Bartonella and Lyme)
COINFECTIONS: A SYNOPSIS
Compiled By: Melanie Reber
Babesiosis
Explanation: Protozoa that invade, infect, and kill the red blood cells
Symptoms: Fatigue, night sweats, fever, chills, weakness, weight loss, nausea, abdominal pain, diarrhea, cough, shortness of breath, headache, neck and back stiffness, dark urine or blood in urine
Treatment: Atovaquone (Mepron) plus Azithromycin (Zithromax), Clindamycin and oral Quinine
Other: Alternative treatment may include Riamet or ArtemisininBartonella
Explanation: Bartonella spp. bacterium
Symptoms: Fever, chills, headache and severe pain in the tibia, weight loss, sore throat, papular or angiomatous rash,burning feet
Treatment: Erythromycin, plus a Fluoroquinolone or RifampinEhrlichiosis (HGE and HME)
Explanation: Rickettsiae that infect the white blood cells
Symptoms: Anemia, fever, chills, headache, muscle pain, rigors, gastrointestinal symptoms, anorexia, fatigue
Treatment: Doxycycline, RifampinRocky Mountain spotted feverii
Explanation: Rickettsia rickettsii parasite that invades the cells lining the heart and blood vessels
Symptoms: High fever, severe headache (especially behind the eyes), maculopapular skin rash
Treatment: Tetracycline, Doxycycline, or ChloramphenicolColorado Tick Fever
Explanation: Reovirus that lodges inside the cells
Symptoms: High fever, chills, severe muscle aches, back pain, headache (especially behind the eyes), light sensitivities, nausea, vomiting, diarrhea
Treatment: No antiviral therapy is available
Other: Aspirin
Relapsing FeverExplanation: Borrelia hermsii spirochete
Symptoms: High fever, sudden chills, eye inflammationi, coughing, jaundice, petechial rash
Treatment: Tetracycline, Doxycycline, or ChloramphenicolTularemia
Explanation: Francisella tularensis bacterium
Symptoms: Painful and swollen lymph nodes, fever, chills, fatigue
Treatment: Tetracycline, ChloramphenicolPowassan encephalitisi
Explanation: Flavivirus that invades and infects the brain
Symptoms: Fever, headache, pain behind the eyes, light sensitivity, muscle weakness, seizures, paralysis, brain inflammation
Treatment: No effective treatmentTick Paralysis
Explanation: A toxic reaction to saliva from female ticks
Symptoms: Paralysis begins in legs and spreads throughout the body within hours
Treatment: Recovery is rapid following the removal of the tickMycoplasma
Explanation: A genus of small bacteria which lack cell walls. M. fermentans, M. pneumoniae, M. penetrans, M. hominis and M. genetalium
Symptoms: Fatigue, headaches, muscle pain and soreness, nausea, gastrointestinal problems, joint pain and soreness, lymph node pain, cognitive problems, depression, breathing problems and other signs and symptoms
Treatment: Slow-growing mycoplasmal infections are not rapidly susceptible to antibioticsi. Doxycycline, Minocycline, Ciprofloxacin, Azithromycin, and Clarithromycin may be used.und heut fühle ich mich so richtig grippig. Interressant!
Es enthält Interleukine, Interferone und Immunglobuline (welche ich merke) und endorphine (von denen ich leider nichts merke)… 🙂
Transfer factor ist Colostrum
Dr. Ryser: The initial reactions to Transfer Factor a patient will experience are similar to a vaccination – but without, of course, exposure to the pathogen. The initial reaction typically includes flu-like symptoms, proportionate to the severity of a patient’s illness. These flu-like symptoms go away, but they prove that the immune system has been activated, and that it is working to suppress the body’s infections.
Furthermore, preliminary studies investigating the serum cytokines in patients receiving transfer factor treatment indicate that the latter activates the Th1 secretion pattern, which has a prognostic value (E. Raise et al., Biotherapy, 9[1-3]:49-54, 1996).
Dr. Ryser: The diagnosis of a patient is of utmost importance. I perform a series of genetic testing with PCR (Polymer Chain Reaction) that tells me the specific bacteria or virus(es) a patient has. Transfer Factor helps with viral, bacterial, and fungal infections as well as parasites, and supports the immune system while treating the problems a patient has. Regarding what formulas of Transfer Factor I use for different patients, I use the plain Transfer Factor as a general prevention treatment, especially for infections and allergies and for patients with Epstein-Barr, Chronic Fatigue Syndrome.
„einige kann man therapieren, oder man kann dann das AB absetzten wenn man den Wirus als Ursache hat.“
Das AB abzusetzen wenn man intrazelluläre erreger zusätzlich hat, wäre nicht so gut. David Wheldon betrachtet die Viren als henchmen. ( Trittbrettfahrer)
Hintergrund ist, das unser Immunsystem besser arbeitet, wenn wir die intrazellulären Erreger eliminiert haben. Ergo sich besser gegen Viren verteidigen kann.
Ich spreche aus Erfahrung, ich habe von Valtrex bis Ribavirin i.V.mit Interferon Alpha + gamma so ziemlich alles versucht um Viren loszuwerden.
Geholfen hats nicht…..
Besser bei viralen Cobelastungen Vit D, Nac, Colostrum, L-Lysin (Herpes&Co) etc. zusätzlich zu ABX.
http://www.davidwheldon.co.uk/hhv6.html
Human Herpes Virus 6, Epstein-Barr Virus
and Endogenous Retroviruses: an input into Multiple Sclerosis?A Brief Note
Human Herpes Virus 6 (HHV6), discovered in 1986, is a very common virus of early childhood, establishing itself in most children around the age of two and often causing a fever lasting for about three days. This age is too early in a person’s life to fit the epidemiological data for the acquisition of a primary pathogen in MS, although rare late primary infection, which might result in chronic active disease, cannot be completely ruled out. The virus is neurotropic, but primary infection seldom causes serious disease. It ordinarily remains dormant in the CNS for life.
Several studies have found elevated indicators of HHV6 in persons with MS. Challoner and co-workers found HHV6 DNA to be very common in the adult human brain; using immunohistochemical methods they found viral protein expression within the nuclei of of oligodendrocytes adjacent to MS lesions. [Challoner PB, Smith KT, Parker JD et al., Plaque-associated expression of human herpesvirus 6 in multiple sclerosis. Proc Natl Acad Sci U S A. 1995 Aug 1;92(16):7440-4.] On the face of it this seems like strong evidence of a primary causal connection. But is it? Let us look at at a parallel situation. Active ongoing infection with HHV6 is generally universal in persons with end-stage AIDS. [Corbellino M, et al. Disseminated human herpesvirus 6 infection in AIDS. Lancet 1993; 342:1242; Knox KK, Carrigan DR. Disseminated active HHV-6 infections in patients with AIDS. Lancet. 1994 Mar 5; 343(8897): 577-8.] The latter workers, using immunohistochemistry, found that in biopsied lymph nodes from AIDS patients the densities of HHV6 infections were significantly (p<0.016) higher in areas undergoing active HIV destruction than in areas free of destructive changes. [http://www.wisconsinlab.com/hiv.htm last viewed 13th Nov 2005.] An increased HHV6 load has been found in the lesions of AIDS-associated Progressive Multifocal Leukoencephalopathy, suggesting a cascade of pathogens (HIV, JC polyoma virus and HHV6.) [Blumberg BM, Mock DJ, Powers JM, et al., The HHV6 paradox: ubiquitous commensal or insidious pathogen? A two-step in situ PCR approach. J Clin Virol. 2000 May; 16(3): 159-78.]
Active HHV6 infection has been found superimposed on Legionnaire’s Disease. [Russler SK, Tapper MA, Knox KK et al., Pneumonitis associated with coinfection by human herpesvirus 6 and Legionella sp. in an immunocompetent adult. Am J Pathol. 1991; 138(6): 1405-11.] 2 of 3 patients with viral encephalitis, in whom both HHV6 and Herpes Simplex Virus (HSV) were isolated, died, a much higher proportion than in encephalitis due to HSV alone, where 1 of 19 died. [Tang YW, Espy MJ, Persing DH, Smith TF. Molecular evidence and clinical significance of herpesvirus coinfection in the central nervous system. J Clin Microbiol. 1997 Nov;35(11):2869-72.]
It seems that HHV6 is what might be termed a ‚henchman‘. That is, an organism widely present in human tissues and which is roused from a state of inactivity to cause gross pathology in areas that are already compromised by a primary intracellular pathogen. (It will be noted that Legionella pneumotropica is a strongly intracellular bacterium.) One might hypothesize that HHV6 may act as a ‚henchman‘ in chronic infections with Chl pneumoniae, which is of course also an intracellular pathogen. One might further speculate that eradication of the chlamydial infection, support of mitochondrial fatigue and restoration of normal immune function might return HHV6 to a state of dormancy.
——————————————————————————–
Epstein-Barr Virus (EBV) is another possible henchman. Usually acquired in childhood, late adolescence, or early adulthood, this common herpesvirus causes infectious mononucleosis and then establishes life-long latency in B lymphocytes; reactivations occur sporadically, with shedding of the virus in the saliva. Antibodies to EBV nuclear antigen-1 slowly rise after infection in the blood, and remain elevated for life. Antibodies to EBVNA-1 are found in oligoclonal bands in the CSF of patients with MS (Rand KH, Houck H et al., Epstein-Barr virus nuclear antigen-1 (EBNA-1) associated oligoclonal bands in patients with multiple sclerosis. J Neurol Sci. 2000 Feb 1;173(1):32-9.) and CD8+ T cell responses to latent EBV proteins are higher in MS patients than in controls. (Sabine Cepok, Dun Zhou et al., Identification of Epstein-Barr virus proteins as putative targets of the immune response in multiple sclerosis. J Clin Invest. 2005 May 2; 115(5): 1352–1360)
——————————————————————————–
Endogenous retroviruses have been suggested as participants in the progression of MS. These ancient sequences of virus DNA are replicas of those inserted into the genome by an accident of infection during germ-cell formation.
Might HERVs play a role in the development of MS? It seems possible. Activation of the HERVs HERV-H/RGH, HERV-W and ERV-9 was described when specific cell types (mainly B cells) from MS patients were cultivated in vitro. Viral RNA from these HERVs has been detected by reverse-transcriptase PCR methods in sera/plasma and brain tissues from MS patients, although not exclusively from these patients [reviewed by Clausen J. Endogenous retroviruses and MS: using ERVs as disease markers. Int MS J. 2003 Apr;10(1):22-8.] Is their activation a cytokine-mediated epiphenomenon, or does it have a pathogenic input into the disease? The many-phased natural history of MS suggests the re-awakening of inherent viruses. Clausen comments: ‚preliminary evidence suggests that specific ERVs may act as auto-, super- or neoantigens with the potential to enhance inflammatory responses or induce autoimmune reactions.‘
Again, might these inner aliens return to a state of dormancy when the primary cause of cytokine disturbance is removed? It seems likely: we have been living with these beings, their genome trapped within ours, for untold time.
Hallo Kundali!
Sehen deine Streifen so aus, dann sind es Bartonellen. Dann Rifa 600 + Atzi 500 für mindestens 3 Monate oder Tavanic(levofloxacin).
http://www.lymediseaseassociation.org/PhotoAlbum_RashBart.html
Bartonellen machen solche hautstreifen
Und noch was ganz wichtiges:
Ich hab so weiße/leichtrosa Streifen in der Hüfte. Die sieht man ganz stark, wenn man die haut strafft. Fühlt sich an wie Einkerbungen, als ob da einfach Gewebe unter der Haut fehlt. Stand das nicht im Zusammenhang mit irgendeiner Erkrankung.Huhu!
Werde vermutlich naechste Woche mit Artemisinin starten.
Da Arte ja über Oxidierung wirkt wollte ich mal fragen welche Antioxidantien man nicht mehr nehmen sollte dann?Also von Q 10 und Vit E weiß ich es zumindest .
Was mit Nac , VitC und Co?
Dankeschön….
Artemesia annua has primarily been used for treatment of protozoan infection. The most active ingredient, artemisinin, is a potent prooxidant whose activity is enhanced by polyunsaturated fats such as cod liver oil and antagonized by vitamin E.
